| CAS No.: | 4478-93-7 |
|---|---|
| Formula: | C6h11nos2 |
| EINECS: | 200-001-8 |
|
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The details of Supply Extract Powder Sulforaphane CAS 4478-93-7 Sulforaphane
| Product Name: | DL-Sulforaphane |
| Synonyms: | sulforafan;sulforaphan;DL-Sulforaphane 4478-93-7;Sulforaphane Racemate;D,L-Sulforaphane - CAS 4478-93-7 - Calbiochem;sulforaphane;R-SULFORAPHANE;R,S-SULFORAPHANE |
| CAS: | 4478-93-7 |
| MF: | C6H11NOS2 |
| MW: | 177.29 |
| EINECS: | 200-001-8 |
| Mol File: | 4478-93-7.mol |
| Boiling point | 125-135°C |
| density | 1.17±0.1 g/cm3(Predicted) |
| storage temp. | -20°C |
| solubility | DMSO: 40 mg/mL, soluble |
| form | liquid |
| color | slightly yellow |
| Stability: | Light Sensitive |
| LogP | 0.407 (est) |
Application of Supply Extract Powder Sulforaphane CAS 4478-93-7 Sulforaphane
Potent, selective inducer of phase II detoxification enzymes with anticarcinogenic properties. Occurs naturally in broccoli. It was found to inhibit chemically induced mammary tumor formation in rats. Antitumor agent
LNCaP prostate cancer cells were treated with DL-Sulforaphane to study the effect on androgen receptor.
Nrf2 activation of the antioxidant response element (ARE) is central to cytoprotective gene expression against oxidative and/or electrophilic stress.
Unless activated by inflammatory, environmental, or oxidative stressors, Nrf2 is sequestered in the cytoplasm by its repressor, Keap1.
Because of its protective capabilities, small molecules that activate Nrf2 signaling are being examined as potential anti-cancer or anti-inflammatory agents.
Sulforaphane is an isothiocyanate derived from cruciferous vegetables, including broccoli, that potently induces chemopreventative enzymes via Keap1-Nrf2 signaling and ARE-driven gene expression.
At 15 μM, sulforaphane inhibits class I and II HDAC activity and suppresses tumor growth by inducing cell cycle arrest and apoptosis selectively in various cancerous prostate epithelial cells without affecting normal cells.
Differential effects of sulforaphane on histone deacetylases, cell cycle arrest and apoptosis in normal prostate cells versus hyperplastic and cancerous prostate cells
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